Smoker's lung is a chronic inflammation of the lungs. Its proper title is Chronic Obstructive Pulmonary Disease (COPD). But this covers several different conditions including those commonly known as chronic bronchitis and emphysema.
Chronic obstructive pulmonary disease (COPD) is a lung disease characterized by inflammation of the lungs and chronically poor airflow escalating over time. Its symptoms include shortness of breath, cough and mucus production and are caused by environmental factors, including exposure to noxious particles. Cigarette smoke is the major cause for COPD, accounting for 80-90% of COPD cases.
The defining feature is obstruction of the flow of air in and out of the lungs which is not completely reversible unlike asthma, where airflow can return to normal and which usually gets progressively worse over time.
Cigarette smoke exposure is a major but preventable cause of increased risk of lung infections in children and adults. Smoke exposure is linked in adults to an increased incidence and severity of asthma, impaired lung function and airway inflammation. Exposure to cigarette smoke has similar adverse effects to active smoking on lung infections, with young children at higher risk, Nearly half of the world's children breathe cigarette smoke at home, and both pre and post-natal smoke exposure are linked to decreased lung function and increased risk and severity of asthma and respiratory infections, Cigarette smoke induces low levels of RelB expression in the lungs, a phenotype that may increase the risk for certain individuals to develop Chronic Obstructive Pulmonary Disease.
Inflammatory markers had a dose-dependent and temporal relationship to smoking and smoking cessation. Both inflammatory and traditional risk factors improved with less smoking, but as the time since smokers quit increased, inflammatory markers resolved more slowly than traditional cardiovascular risk factors. Still, the smoking-associated inflammatory response returned to normal within five years after smokers quit.
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